| Name | L-(+)-Arabinose |
| Description | L-(+)-Arabinose selectively inhibits intestinal sucrase in a non-competitive manner, thereby preventing glucose elevation induced by sucrose intake. |
| In vitro | L-(+)-Arabinose is associated with ribose-5-phosphate isomerase deficiency, an inborn metabolic error[1]. It selectively inhibits intestinal sucrase in a noncompetitive manner, thereby suppressing the increase in plasma glucose following sucrose consumption[1]. |
| In vivo | L-(+)-Arabinose feeding reduced the weights of epididymal adipose tissue in rats. Moreover, plasma insulin and triacylglycerol concentrations were significantly reduced by dietary L-(+)-Arabinose[1]. L-(+)-Arabinose suppresses gluconeogenesis through modulating AMP-activated protein kinase in metabolic disorder mice[2]. |
| Storage | Powder: -20°C for 3 years | In solvent: -80°C for 1 year | Shipping with blue ice. |
| Solubility Information | 10% DMSO+40% PEG300+5% Tween 80+45% Saline : 1 mg/mL (6.66 mM), Sonication is recommended. DMSO : 60 mg/mL (399.65 mM), Sonication is recommended. H2O : 400 mg/mL (2664.36 mM), Sonication is recommended.
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| Keywords | L-Arabinose | L(+)Arabinose | L (+) Arabinose | Inhibitor | inhibit | Human Endogenous Metabolite | EndogenousMetabolite | Endogenous Metabolite |
| Inhibitors Related | Sucrose | Aceglutamide | Nicotinamide riboside malate | DL-Lysine | D(+)-Raffinose pentahydrate | Guanidine hydrochloride | Malic acid | Formamide | Glycerol | Thymidine | Corn starch | Gluconate Calcium |
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